Potentized homoeopathic drugs act through regulation of geneexpression: a hypothesis to explain their mechanism and pathways of action in vitro

Abstract: A working hypothesis to explain the mechanism of action of potentized homoeopathic drugs in vivo has been proposed. The model is partly substantiated from our own research data on repair of chromosomal damages in X-irradiated or toxic chemical-treated mice by the oral administration of some potentized homoeopathic drugs, and partly from some of the unpublished and published works of other researchers in the field of homoeopathy. In this model, strong scientific arguments have been made to form the hypothesis that the potentized homoeopathic drugs act through regulation of gene-expression, presumably through hormone hormone-protein complexes the sensorgene-integrator gene-receptor gene-producer gene pathway of Britten and Davidson's model, or else through the regulator/mutator gene-operator gene-structural gene pathway of Jacob and Monod's model among some other independent mechanisms. Scientific details of some possible pathways, admittedly speculative for some steps, have also been provided to stimulate research in this direction to verify the correctness of the hypothesis.

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